Publikationen in Zusammenarbeit mit Forschern von Instituto de Biología Molecular y Celular del Cáncer de Salamanca / Centro de Investigación del Cáncer (21)

2022

  1. Transient Inhibition of the JAK/STAT Pathway Prevents B-ALL Development in Genetically Predisposed Mice

    Cancer research, Vol. 82, Núm. 6, pp. 1098-1109

2020

  1. An intact gut microbiome protects genetically predisposed mice against leukemia

    Blood, Vol. 136, Núm. 18, pp. 2003-2017

  2. Inhibition of inflammatory signaling in Pax5 mutant cells mitigates B-cell leukemogenesis

    Scientific Reports, Vol. 10, Núm. 1

  3. Pathophysiological mechanisms underlying a rat model of triple whammy acute kidney injury

    Laboratory Investigation, Vol. 100, Núm. 11, pp. 1455-1464

2019

  1. Dnmt1 links BCR-ABLp210 to epigenetic tumor stem cell priming in myeloid leukemia

    Leukemia, Vol. 33, Núm. 1, pp. 249-278

  2. Infectious stimuli promote malignant B-cell acute lymphoblastic leukemia in the absence of AID

    Nature Communications, Vol. 10, Núm. 1

2018

  1. Lmo2 expression defines tumor cell identity during T-cell leukemogenesis

    EMBO Journal, Vol. 37, Núm. 14

  2. Loss of Pax5 exploits sca1-BCR-ABLp190 susceptibility to confer the metabolic shift essential for pB-ALL

    Cancer Research, Vol. 78, Núm. 10, pp. 2669-2679

  3. Supplementary data for the biological age linked to oxidative stress modifies breast cancer aggressiveness

    Data in Brief

2017

  1. Infection exposure promotes ETV6-RUNX1 precursor B-cell leukemia via impaired H3K4 demethylases

    Cancer Research, Vol. 77, Núm. 16, pp. 4365-4377

2015

  1. Immunosuppression-Independent Role of Regulatory T Cells against Hypertension-Driven Renal Dysfunctions

    Molecular and Cellular Biology, Vol. 35, Núm. 20, pp. 3528-3546

  2. Infection exposure is a causal factor in B-cell precursor acute lymphoblastic leukemia as a result of Pax5-inherited susceptibility

    Cancer Discovery, Vol. 5, Núm. 12, pp. 1328-1343

  3. Unraveling heterogeneous susceptibility and the evolution of breast cancer using a systems biology approach

    Genome Biology, Vol. 16, Núm. 1

2012

  1. A novel molecular mechanism involved in multiple myeloma development revealed by targeting MafB to haematopoietic progenitors

    EMBO Journal, Vol. 31, Núm. 18, pp. 3704-3717

  2. Oxysterol-induced soluble endoglin release and its involvement in hypertension

    Circulation, Vol. 126, Núm. 22, pp. 2612-2624

2010

  1. Cancer as a reprogramming-like disease: Implications in tumor development and treatment

    Seminars in Cancer Biology, Vol. 20, Núm. 2, pp. 93-97

  2. Deletion of H-Ras decreases renal fibrosis and myofibroblast activation following ureteral obstruction in mice

    Kidney International, Vol. 77, Núm. 6, pp. 509-518

2009

  1. Targeted genomic disruption of H-ras and N-ras has no effect on early renal changes after unilateral ureteral ligation

    World Journal of Urology, Vol. 27, Núm. 6, pp. 787-797

2008

  1. Involvement of small Ras GTPases and their effectors in chronic renal disease

    Cellular and Molecular Life Sciences, Vol. 65, Núm. 3, pp. 477-492

2006

  1. TGF-β1 induces COX-2 expression and PGE2 synthesis through MAPK and PI3K pathways in human mesangial cells

    Kidney International, Vol. 70, Núm. 5, pp. 901-909