Iván
Quesada Moll
Publicaciones en las que colabora con Iván Quesada Moll (13)
2022
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Insulin-degrading enzyme ablation in mouse pancreatic alpha cells triggers cell proliferation, hyperplasia and glucagon secretion dysregulation
Diabetologia, Vol. 65, Núm. 8, pp. 1375-1389
2019
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Cortistatin regulates glucose-induced electrical activity and insulin secretion in mouse pancreatic beta-cells
Molecular and Cellular Endocrinology, Vol. 479, pp. 123-132
2016
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Maternal exposure to bisphenol-A during pregnancy increases pancreatic β-cell growth during early life in male mice offspring
Endocrinology, Vol. 157, Núm. 11, pp. 4158-4171
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The bile acid TUDCA increases glucose-induced insulin secretion via the cAMP/PKA pathway in pancreatic beta cells
Metabolism: Clinical and Experimental, Vol. 65, Núm. 3, pp. 54-63
2015
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Glucagon increases beating rate but not contractility in rat right atrium. comparison with isoproterenol
PLoS ONE, Vol. 10, Núm. 7
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Pancreatic alpha-cells from female mice undergo morphofunctional changes during compensatory adaptations of the endocrine pancreas to diet-induced obesity
Scientific Reports, Vol. 5
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Pancreatic α Cells are Resistant to Metabolic Stress-induced Apoptosis in Type 2 Diabetes
EBioMedicine, Vol. 2, Núm. 5, pp. 378-385
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Role of the clock gene Rev-erbα in metabolism and in the endocrine pancreas
Diabetes, Obesity and Metabolism, Vol. 17, Núm. S1, pp. 106-114
2014
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Nutrient regulation of glucagon secretion: Involvement in metabolism and diabetes
Nutrition Research Reviews, Vol. 27, Núm. 1, pp. 48-62
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Pancreatic alpha-cell dysfunction contributes to the disruption of glucose homeostasis and compensatory insulin hypersecretion in glucocorticoid-treated rats
PLoS ONE, Vol. 9, Núm. 4
2013
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Insulin hypersecretion in islets from diet-induced hyperinsulinemic obese female mice is associated with several functional adaptations in individual β-cells
Endocrinology, Vol. 154, Núm. 10, pp. 3515-3524
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Involvement of the Clock Gene Rev-erb alpha in the Regulation of Glucagon Secretion in Pancreatic Alpha-Cells
PLoS ONE, Vol. 8, Núm. 7