Publikationen in Zusammenarbeit mit Forschern von Instituto de Salud Carlos III (12)

2023

  1. Identification of Pre-Renal and Intrinsic Acute Kidney Injury by Anamnestic and Biochemical Criteria: Distinct Association with Urinary Injury Biomarkers

    International journal of molecular sciences, Vol. 24, Núm. 3

  2. Renal sympathetic activity: A key modulator of pressure natriuresis in hypertension

    Biochemical Pharmacology, Vol. 208

2022

  1. Neural Network-Based Calculator for Rat Glomerular Filtration Rate

    Biomedicines, Vol. 10, Núm. 3

2021

  1. Albuminuria pre-emptively identifies cardiac patients at risk of contrast-induced nephropathy

    Journal of Clinical Medicine, Vol. 10, Núm. 21

  2. Haemodynamic frailty – A risk factor for acute kidney injury in the elderly

    Ageing Research Reviews, Vol. 70

  3. The furosemide stress test and computational modeling identify renal damage sites associated with predisposition to acute kidney injury in rats

    Translational Research, Vol. 231, pp. 76-91

2020

  1. A meta-analysis of preclinical studies using antioxidants for the prevention of cisplatin nephrotoxicity: implications for clinical application

    Critical Reviews in Toxicology, Vol. 50, Núm. 9, pp. 780-800

  2. Combined use of GM2AP and TCP1-eta urinary levels predicts recovery from intrinsic acute kidney injury

    Scientific Reports, Vol. 10, Núm. 1

  3. Impaired Tubular Reabsorption Is the Main Mechanism Explaining Increases in Urinary NGAL Excretion Following Acute Kidney Injury in Rats

    Toxicological sciences : an official journal of the Society of Toxicology, Vol. 175, Núm. 1, pp. 75-86

  4. Pathophysiological mechanisms underlying a rat model of triple whammy acute kidney injury

    Laboratory Investigation, Vol. 100, Núm. 11, pp. 1455-1464

  5. Urinary TCP1-eta: A Cortical Damage Marker for the Pathophysiological Diagnosis and Prognosis of Acute Kidney Injury

    Toxicological Sciences, Vol. 174, Núm. 1, pp. 3-15

  6. Urinary transferrin pre-emptively identifies the risk of renal damage posed by subclinical tubular alterations

    Biomedicine and Pharmacotherapy, Vol. 121