Publicacións nas que colabora con Ana Purificación Velasco Criado (14)
2018
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Endocytosis and Transcytosis of Amyloid-β Peptides by Astrocytes: A Possible Mechanism for Amyloid-β Clearance in Alzheimer's Disease
Journal of Alzheimer's Disease, Vol. 65, Núm. 4, pp. 1109-1124
2017
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Restrained Phosphatidylcholine Synthesis in a Cellular Model of Down’s Syndrome is Associated with the Overexpression of Dyrk1A
Molecular Neurobiology, Vol. 54, Núm. 2, pp. 1092-1100
2016
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Aberrant co-localization of synaptic proteins promoted by Alzheimer's Disease amyloid-β peptides: Protective effect of human serum albumin
Journal of Alzheimer's Disease, Vol. 55, Núm. 1, pp. 171-182
2013
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Overexpression of DYRK1A inhibits choline acetyltransferase induction by oleic acid in cellular models of Down syndrome
Experimental Neurology, Vol. 239, Núm. 1, pp. 229-234
2012
2009
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Albumin endocytosis via megalin in astrocytes is caveola- and Dab-1 dependent and is required for the synthesis of the neurotrophic factor oleic acid
Journal of Neurochemistry, Vol. 111, Núm. 1, pp. 49-60
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La megalina es el receptor para la endocitosis mediada por caveolas de la albúmina y se requiere para la síntesis del factor neurotrófico ácido oleico
Trauma, Vol. 20, Núm. 3, pp. 130-136
2008
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Megalin is a receptor for albumin in astrocytes and is required for the synthesis of the neurotrophic factor oleic acid
Journal of Neurochemistry, Vol. 106, Núm. 3, pp. 1149-1159
2004
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The neurotrophic effect of oleic acid includes dendritic differentiation and the expression of the neuronal basic helix-loop-helix transcription factor NeuroD2
Journal of Neurochemistry, Vol. 88, Núm. 5, pp. 1041-1051
2003
2002
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Transcytosis of albumin in astrocytes activates the sterol regulatory element-binding protein-1, which promotes the synthesis of the neurotrophic factor oleic acid
Journal of Biological Chemistry, Vol. 277, Núm. 6, pp. 4240-4246
2001
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Neuronal differentiation is triggered by oleic acid synthesized and released by astrocytes
Journal of Neurochemistry, Vol. 79, Núm. 3, pp. 606-616
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The enhancement of glucose uptake caused by the collapse of gap junction communication is due to an increase in astrocyte proliferation
Journal of Neurochemistry, Vol. 78, Núm. 4, pp. 890-898
2000
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ATP-Sensitive potassium channel regulates astrocytic gap junction permeability by a Ca2+-independent mechanism
Journal of Neurochemistry, Vol. 74, Núm. 3, pp. 1249-1256