Publicacións (347) Publicacións de José Miguel López Novoa

2022

  1. Functional Alterations Involved in Increased Bleeding in Hereditary Hemorrhagic Telangiectasia Mouse Models

    Frontiers in Medicine, Vol. 9

2021

  1. El sino de los Rebolledo

    Ediciones Atlantis

  2. Pregnancy-induced high plasma levels of soluble endoglin in mice lead to preeclampsia symptoms and placental abnormalities

    International Journal of Molecular Sciences, Vol. 22, Núm. 1, pp. 1-20

2020

  1. Evocaciones: hoguera de amor y llama de olvido

    2020

  2. Impaired Tubular Reabsorption Is the Main Mechanism Explaining Increases in Urinary NGAL Excretion Following Acute Kidney Injury in Rats

    Toxicological sciences : an official journal of the Society of Toxicology, Vol. 175, Núm. 1, pp. 75-86

  3. Potential Role of Circulating Endoglin in Hypertension via the Upregulated Expression of BMP4

    Cells, Vol. 9, Núm. 4

2019

  1. Preventive effect of cardiotrophin-1 administration before DSS-induced ulcerative colitis in mice

    Journal of Clinical Medicine, Vol. 8, Núm. 12

2018

  1. Pequeñas personas peculiares

    PiEdiciones

  2. Why do we age? theories and practical consequences

    Revista Electrónica de Biomedicina, Núm. 2, pp. 59-64

2015

  1. Acute tubular necrosis: An old term in search for a new meaning within the evolving concept of acute kidney injury

    New Horizons in Translational Medicine, Vol. 2, Núm. 4-5, pp. 110-117

  2. High soluble endoglin levels do not induce endothelial dysfunction in mouse aorta

    PLoS ONE, Vol. 10, Núm. 3

  3. Importance of the understanding the mechanisms involved in post-ischemic revascularization in vascular surgery

    Angiologia, Vol. 67, Núm. 3, pp. 163-164

  4. Plasma cardiotrophin-1 as a marker of hypertension and diabetes-induced target organ damage and cardiovascular risk

    Medicine (United States), Vol. 94, Núm. 30

  5. Snail1-induced partial epithelial-to-mesenchymal transition drives renal fibrosis in mice and can be targeted to reverse established disease

    Nature Medicine, Vol. 21, Núm. 9, pp. 989-997

  6. Sub-nephrotoxic cisplatin sensitizes rats to acute renal failure and increases urinary excretion of fumarylacetoacetase

    Toxicology Letters, Vol. 234, Núm. 2, pp. 99-109

  7. Translational value of animal models of kidney failure

    European Journal of Pharmacology, Vol. 759, pp. 205-220

  8. Urinary proteomics in renal pathophysiology: Impact of proteinuria

    Proteomics - Clinical Applications, Vol. 9, Núm. 5-6, pp. 636-640

2014

  1. Endoglin involvement 1 in integrin-mediated cell adhesion as a putative pathogenic mechanism in Hereditary Hemorrhagic Telangectasia type 1 (HHT1)

    Frontiers in Genetics, Vol. 5, Núm. DEC

  2. Impaired wound repair in adult endoglin heterozygous mice associated with lower NO bioavailability

    Journal of Investigative Dermatology, Vol. 134, Núm. 1, pp. 247-255

  3. Interferon-y Reduces the Proliferation of Primed Human Renal Tubular Cells

    Nephron Extra, Vol. 4, Núm. 1, pp. 1-7