Hospital Universitario de Salamanca -ko ikertzaileekin lankidetzan egindako argitalpenak (8)

2016

  1. Mechanisms of triple whammy acute kidney injury

    Pharmacology and Therapeutics, Vol. 167, pp. 132-145

2015

  1. Activation of the ALK-5 pathway is not per se sufficient for the antiproliferative effect of TGF-β1 on renal tubule epithelial cells

    Cellular Physiology and Biochemistry, Vol. 37, Núm. 4, pp. 1231-1239

  2. Increased Klk9 urinary excretion is associated to hypertension-induced cardiovascular damage and renal alterations

    Medicine (United States), Vol. 94, Núm. 41

2012

  1. Subcellular targets of cisplatin cytotoxicity: An integrated view

    Pharmacology and Therapeutics, Vol. 136, Núm. 1, pp. 35-55

2011

  1. Necrotic concentrations of cisplatin activate the apoptotic machinery but inhibit effector caspases and interfere with the execution of apoptosis

    Toxicological Sciences, Vol. 122, Núm. 1, pp. 73-85

  2. Urinary levels of regenerating islet-derived protein III Β and gelsolin differentiate gentamicin from cisplatin-induced acute kidney injury in rats

    Kidney International, Vol. 79, Núm. 5, pp. 518-528

2010

  1. Apoptotic volume decrease as a geometric determinant for cell dismantling into apoptotic bodies

    Cell Death and Differentiation, Vol. 17, Núm. 11, pp. 1665-1671

  2. Sub-nephrotoxic doses of gentamicin predispose animals to developing acute kidney injury and to excrete ganglioside M2 activator protein

    Kidney International, Vol. 78, Núm. 10, pp. 1006-1015