Publikationen in Zusammenarbeit mit Forschern von Instituto de Investigación Biomédica de Salamanca (18)

2024

  1. Urinary GM2AP coincides with renal cortical damage and grades cisplatin nephrotoxicity severity in rats

    Toxicology, Vol. 508

2023

  1. Identification of Pre-Renal and Intrinsic Acute Kidney Injury by Anamnestic and Biochemical Criteria: Distinct Association with Urinary Injury Biomarkers

    International journal of molecular sciences, Vol. 24, Núm. 3

  2. Pathophysiology of Acute Kidney Frailty

    Physiology (Bethesda, Md.), Vol. 38, Núm. 6, pp. 0

2022

  1. Diagnosis of Cardiac Surgery-Associated Acute Kidney Injury: State of the Art and Perspectives

    Journal of Clinical Medicine, Vol. 11, Núm. 15

  2. The Urinary Level of Injury Biomarkers Is Not Univocally Reflective of the Extent of Toxic Renal Tubular Injury in Rats

    International Journal of Molecular Sciences, Vol. 23, Núm. 7

  3. Urinary KIM-1 Correlates with the Subclinical Sequelae of Tubular Damage Persisting after the Apparent Functional Recovery from Intrinsic Acute Kidney Injury

    Biomedicines, Vol. 10, Núm. 5

2021

  1. Biomarkers of persistent renal vulnerability after acute kidney injury recovery

    Scientific Reports, Vol. 11, Núm. 1

  2. Urinary Plasminogen Activator Inhibitor-1: A Biomarker of Acute Tubular Injury

    American Journal of Nephrology, Vol. 52, Núm. 9, pp. 714-724

2020

  1. Combined use of GM2AP and TCP1-eta urinary levels predicts recovery from intrinsic acute kidney injury

    Scientific Reports, Vol. 10, Núm. 1

  2. Impaired Tubular Reabsorption Is the Main Mechanism Explaining Increases in Urinary NGAL Excretion Following Acute Kidney Injury in Rats

    Toxicological sciences : an official journal of the Society of Toxicology, Vol. 175, Núm. 1, pp. 75-86

  3. Pathophysiological mechanisms underlying a rat model of triple whammy acute kidney injury

    Laboratory Investigation, Vol. 100, Núm. 11, pp. 1455-1464

  4. Urinary TCP1-eta: A Cortical Damage Marker for the Pathophysiological Diagnosis and Prognosis of Acute Kidney Injury

    Toxicological Sciences, Vol. 174, Núm. 1, pp. 3-15

2018

  1. N-acetylcysteine transforms necrosis into apoptosis and affords tailored protection from cisplatin cytotoxicity

    Toxicology and Applied Pharmacology, Vol. 349, pp. 83-93

2015

  1. Activation of the ALK-5 pathway is not per se sufficient for the antiproliferative effect of TGF-β1 on renal tubule epithelial cells

    Cellular Physiology and Biochemistry, Vol. 37, Núm. 4, pp. 1231-1239

  2. Acute tubular necrosis: An old term in search for a new meaning within the evolving concept of acute kidney injury

    New Horizons in Translational Medicine, Vol. 2, Núm. 4-5, pp. 110-117

  3. Increased Klk9 urinary excretion is associated to hypertension-induced cardiovascular damage and renal alterations

    Medicine (United States), Vol. 94, Núm. 41

  4. Pathophysiological role of different tubular epithelial cell death modes in acute kidney injury

    Clinical Kidney Journal, Vol. 8, Núm. 5, pp. 548-559

2012

  1. Subcellular targets of cisplatin cytotoxicity: An integrated view

    Pharmacology and Therapeutics, Vol. 136, Núm. 1, pp. 35-55