University of Pittsburgh School of Medicine-ko ikertzaileekin lankidetzan egindako argitalpenak (9)

2009

  1. Mutant parathyroid hormone-related protein, devoid of the nuclear localization signal, markedly inhibits arterial smooth muscle cell cycle and neointima formation by coordinate up-regulation of p15 Ink4b and p27 kip1

    Endocrinology, Vol. 150, Núm. 3, pp. 1429-1439

2007

  1. Tissue-specific deletion of the retinoblastoma protein in the pancreatic β-cell has limited effects on β-cell replication, mass, and function

    Diabetes, Vol. 56, Núm. 1, pp. 57-64

2006

  1. Cellular mechanism through which parathyroid hormone-related protein induces proliferation in arterial smooth muscle cells: Definition of an arterial smooth muscle PTHrP/p27kip1 pathway

    Circulation Research, Vol. 99, Núm. 9, pp. 933-942

  2. Evaluation of β-cell replication in mice transgenic for hepatocyte growth factor and placental lactogen: Comprehensive characterization of the G1/S regulatory proteins reveals unique involvement of p21 cip

    Diabetes, Vol. 55, Núm. 1, pp. 70-77

  3. Molecular control of cell cycle progression in the pancreatic β-cell

    Endocrine Reviews, Vol. 27, Núm. 4, pp. 356-370

  4. The cell cycle inhibitory protein p21cip is not essential for maintaining β-cell cycle arrest or β-cell function in vivo

    Diabetes, Vol. 55, Núm. 12, pp. 3271-3278

2005

  1. Molecular engineering human hepatocytes into pancreatic beta cells for diabetes therapy

    Proceedings of the National Academy of Sciences of the United States of America

2004

  1. Hepatocyte Growth Factor Gene Therapy for Pancreatic Islets in Diabetes: Reducing the Minimal Islet Transplant Mass Required in a Glucocorticoid-Free Rat Model of Allogeneic Portal Vein Islet Transplantation

    Endocrinology, Vol. 145, Núm. 2, pp. 467-474

  2. Induction of β-Cell Proliferation and Retinoblastoma Protein Phosphorylation in Rat and Human Islets Using Adenovirus-Mediated Transfer of Cyclin-Dependent Kinase-4 and Cyclin D1

    Diabetes, Vol. 53, Núm. 1, pp. 149-159