Publicaciones en colaboración con investigadores/as de Centro de Investigación Biomédica en Red sobre Enfermedades Hepáticas y Digestivas (22)

2024

  1. Idiosyncratic Drug-Induced Liver Injury and Amoxicillin–Clavulanate: Spotlight on Gut Microbiota, Fecal Metabolome and Bile Acid Profile in Patients

    International Journal of Molecular Sciences, Vol. 25, Núm. 13

2022

  1. Effect of COVID-19 on quality of life of persons aged >70 years with adult spinal deformity: A cross-sectional case-control study

    Medicine, Vol. 101, Núm. 33, pp. e29954

2021

  1. Aging, gut microbiota and metabolic diseases: Management through physical exercise and nutritional interventions

    Nutrients, Vol. 13, Núm. 1, pp. 1-21

  2. Long-term effects of bariatric surgery on gut microbiota composition and faecal metabolome related to obesity remission

    Nutrients, Vol. 13, Núm. 8

  3. Molecular mechanisms of hepatotoxic cholestasis by clavulanic acid: Role of NRF2 and FXR pathways

    Food and Chemical Toxicology, Vol. 158

  4. The synbiotic combination of akkermansia muciniphila and quercetin ameliorates early obesity and NAFLD through gut microbiota reshaping and bile acid metabolism modulation

    Antioxidants, Vol. 10, Núm. 12

2020

  1. Exercise training modulates the gut microbiota profile and impairs inflammatory signaling pathways in obese children

    Experimental and Molecular Medicine, Vol. 52, Núm. 7, pp. 1048-1061

2019

  1. A Network Involving Gut Microbiota, Circulating Bile Acids, and Hepatic Metabolism Genes That Protects Against Non-Alcoholic Fatty Liver Disease

    Molecular Nutrition and Food Research, Vol. 63, Núm. 20

  2. Beneficial effects of exercise on gut microbiota functionality and barrier integrity, and gut-liver crosstalk in an in vivo model of early obesity and non-alcoholic fatty liver disease

    DMM Disease Models and Mechanisms, Vol. 12, Núm. 5

  3. Functional Interactions between Gut Microbiota Transplantation, Quercetin, and High-Fat Diet Determine Non-Alcoholic Fatty Liver Disease Development in Germ-Free Mice

    Molecular Nutrition and Food Research, Vol. 63, Núm. 8

2018

  1. Autophagy as a molecular target of flavonoids underlying their protective effects in human disease

    Current Medicinal Chemistry, Vol. 25, Núm. 7, pp. 814-838

  2. Intestinal microbiota modulation in obesity-related non-alcoholic fatty liver disease

    Frontiers in Physiology, Vol. 9

2017

  1. Protective effect of quercetin on high-fat diet-induced non-alcoholic fatty liver disease in mice is mediated by modulating intestinal microbiota imbalance and related gut-liver axis activation

    Free Radical Biology and Medicine, Vol. 102, pp. 188-202

2016

  1. Hepatocyte vitamin D receptor regulates lipid metabolism and mediates experimental diet-induced steatosis

    Journal of Hepatology, Vol. 65, Núm. 4, pp. 748-757

2015

  1. Flavonoids and related compounds in non-alcoholic fatty liver disease therapy

    Current Medicinal Chemistry, Vol. 22, Núm. 25, pp. 2991-3012

  2. Quercetin ameliorates dysregulation of lipid metabolism genes via the PI3K/AKT pathway in a diet-induced mouse model of nonalcoholic fatty liver disease

    Molecular Nutrition and Food Research, Vol. 59, Núm. 5, pp. 879-893

  3. Repression of the nuclear receptor small heterodimer partner by steatotic drugs and in advanced nonalcoholic fatty liver disease

    Molecular Pharmacology, Vol. 87, Núm. 4, pp. 582-594

2014

  1. Modulation of PI3K-LXRα-dependent lipogenesis mediated by oxidative/nitrosative stress contributes to inhibition of HCV replication by quercetin

    Laboratory Investigation, Vol. 94, Núm. 3, pp. 262-274

2013

  1. The human liver fatty acid binding protein (FABP1) gene is activated by FOXA1 and PPARα; And repressed by C/EBPα: Implications in FABP1 down-regulation in nonalcoholic fatty liver disease

    Biochimica et Biophysica Acta - Molecular and Cell Biology of Lipids, Vol. 1831, Núm. 4, pp. 803-818

2012

  1. Liver X receptor α-mediated regulation of lipogenesis by core and NS5A proteins contributes to HCV-induced liver steatosis and HCV replication

    Laboratory Investigation, Vol. 92, Núm. 8, pp. 1191-1202