Alberto Martín Lorenzo-rekin lankidetzan egindako argitalpenak (12)

2018

  1. Lmo2 expression defines tumor cell identity during T-cell leukemogenesis

    EMBO Journal, Vol. 37, Núm. 14

  2. Loss of Pax5 exploits sca1-BCR-ABLp190 susceptibility to confer the metabolic shift essential for pB-ALL

    Cancer Research, Vol. 78, Núm. 10, pp. 2669-2679

2017

  1. Activation-induced cytidine deaminase prevents pro-B cell acute lymphoblastic leukemia by functioning as a negative regulator in Rag1 deficient pro-B cells

    Oncotarget, Vol. 8, Núm. 44, pp. 75797-75807

  2. Crebbp loss cooperates with Bcl2 overexpression to promote lymphoma in mice

    Blood, Vol. 129, Núm. 19, pp. 2645-2656

  3. Infection exposure promotes ETV6-RUNX1 precursor B-cell leukemia via impaired H3K4 demethylases

    Cancer Research, Vol. 77, Núm. 16, pp. 4365-4377

  4. Prolonged intracellular accumulation of light-inducible nanoparticles in leukemia cells allows their remote activation

    Nature Communications, Vol. 8

2016

  1. Could vitamin d analogues be used to target leukemia stem cells?

    International Journal of Molecular Sciences, Vol. 17, Núm. 6

2015

  1. Infection causes childhood leukemia

    Aging

  2. Infection exposure is a causal factor in B-cell precursor acute lymphoblastic leukemia as a result of Pax5-inherited susceptibility

    Cancer Discovery, Vol. 5, Núm. 12, pp. 1328-1343

2014

  1. Cancer Stem Cell as a Result of a Reprogramming-Like Mechanism: Implications in Tumor Development and Treatment

    Cancer Stem Cells (Wiley Blackwell), pp. 61-77

  2. Early epigenetic cancer decisions

    Biological Chemistry, Vol. 395, Núm. 11, pp. 1315-1320

2013

  1. Genetic background affects susceptibility to tumoral stem cell reprogramming

    Cell Cycle, Vol. 12, Núm. 15, pp. 2505-2509